[3] Phan TT, Lim IJ, Chan SY, et al. Suppression of transforming growth factor beta/smad signaling in keloid-derived fibroblasts by quercetin implications for the treatment of excessive scars[J]. J Trauma,2004,57(5):1032-1037.
[4] Lee SF,Lin JK. Inhibitory effects of phytopolyphenolson TPA-induced transformation, PKC activation, and c-jun expression in mouse fibroblast cells[J]. Nytr Cancer,1998,28(2):177-183
[5] Phan TT, See P, Tran E, et al. Suppression of insulin-like growth factor signalling pathway and collagen expression in keloid-derived fibroblasts by quercetin: its therapeutic potential use in the treatment and/or prevention of keloids[J]. Br J Dermatol,2003,148(3):544-552.
[6] Phan TT, Lim IJ, Sun L, et al. Quercetin inhibits fibronectin production by keloid-derived fibroblasts. Implication for the treatment of excessive scars[J]. J Dermatol Sci,2003,33(3):192-194.
[7] Cordeiro MF,Reichel MB,et al. TGF-β1,β2,β3 in vivo:effect on normal and mitomycin C-modulated conjunctival scar-ring[J]. Invest Ophalmol Vis Sci,1999,40(9):1975-7982.
[8] Rossert J, Terraz-Durasnel C, Brideau G. Growth factors,cytokines, and renal fibrosis during the course of diabetic nephropathy[J]. Diabetes Metab,2000,26(Suppl 4):16-24.